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ECG Case 9

12/15/2012 by Adam Thompson, EMT-P 1 Comment
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You respond to a nursing home for a 65 year-old female complaining of left arm pain. The patient looks pale and is cool and clammy. She denies chest pain but has been experiencing exertional dyspnea.You obtain the 12-lead below. What is your interpretation and what treatments would you provide?

ECG Case 9 – Click to enlarge

 


 

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Filed Under: Assessment, Cardiology, Case Reviews, Clinical Discussion, ECG/EKG Archive, Medical Emergencies, paramedics

ECG Case 8

12/01/2012 by Adam Thompson, EMT-P 1 Comment
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You respond to a residence for a 24 year-old male with apparent anxiety. He is hyperventilating and tells you that he feels really anxious. He has no other complaints.S – Shortness of breath and parasthesia to both hands.

A – NKDA
M – Adderall
P – ADHD and depression
L – Tells you he just had a sandwich an hour ago.
E – Says he was studying for college finals.Resp. Rate – 40 breaths/min.
HR – 115 beats/min,
BP – 140/90 mmHg
O2 Sat – 100%
Skin – Pink/Warm/Dry

ECG Case 8 – Click to enlarge

 


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Filed Under: Assessment, Cardiology, Case Reviews, Clinical Discussion, ECG/EKG Archive, Education, Medical Emergencies

ECG Case 7

11/14/2012 by Adam Thompson, EMT-P 1 Comment
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You respond to a 59 y/o male at his residence. He is complaining of severe chest pain and shortness of breath. The patient is pale, cool, and diaphoretic. Here is his ECG, what do you think?

ECG Case 7 – Click to enlarge

 

 


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Filed Under: Cardiology, Case Reviews, Clinical Discussion, Critical Judgment, ECG/EKG Archive, Education, Medical Emergencies, paramedics

ECG Case 5

11/05/2012 by Adam Thompson, EMT-P 2 Comments
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You respond to a 65 y/o Male at his residence. His daughter, on scene, called 911 because she is worried about her father’s health. She states that he just hasn’t been acting right. “He is weaker than normal, and becomes short of breath very easily”. The patient himself is not thrilled about your presence. He is a rather obese man (about 400 lbs), and he is sitting in his recliner sans shirt or pants. His immediate area provides evidence that he doesn’t move

 from that chair very often.

S – The patient states that he is always weak and it is normal for him to get short of breath when he gets up.
A – NKDA
M – Glucophage, Gabapentin, Albuterol, Singulair, Prevacid, Carevedilol, Enalapril, Digoxin, Aspirin, Oxygen
P – AMI, CHF, Asthma, Non-insulin dependent diabetes, AICD
L – Oreos and Orange Juice
E – Sitting in his chair

B/P: 61/37, Left Arm
SpO2: 83, on 2 lpm O2,
Pulse: 40 & regular
Resp: 30 & regular
Skin: Pale, cool, & clammy

You place your patient on the monitor and obtain the following 12-lead. What would you immediately ask your patient? What is your interpretation of the ECG? What treatments would you provide?

ECG 5 – Click to enlarge

 


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Filed Under: Cardiology, Case Reviews, Clinical Discussion, ECG/EKG Archive, Education, Medical Emergencies, paramedics, Toxicology

ECG: 40 y/o Female with Chest Pain

08/12/2010 by Adam Thompson, EMT-P 24 Comments
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Also Posted over at ECG Experts

Submission thanks to Tim Waters, CCEMTP of Lee County Medstar

40 yo female, thin build with no history/meds/allergies. + smoker. Works as painter outside and was painting when developed pain in her upper chest/left arm which is the same she uses to paint. Also adds that she has been moving and lifting numerous heavy objects over the past week and since then has been having these episodes of shoulder discomfort. Pain is non-radiating with moderate reproducibility with movement and inspiration. I forget what severity scale she gave it but was definitely uncomfortable. Onset was about 3 ½ hours prior to presentation while painting with her trying to work through the pain until it became to unbearable.. Denies nausea, is diaphoretic but has been working outside.

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Filed Under: Case Reviews, ECG/EKG Archive

When All You Have is a Hammer, Every Problem Looks Like… Lasix?

06/22/2010 by Adam Thompson, EMT-P 1 Comment
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I posted this article over at http://www.lifeunderthelights.com/ a few days ago and I thought it would good for here as well. I’m sorry that I’ve only rarely posted here, but I read this blog quite often and recommend it to all of my peers. Enjoy

———————————————–

A few years ago I responded to a structure fire on the main engine out of my station. The fire was at a house that had been converted to a dog kennel and grooming shop just a few blocks away from the firehouse and was a short response time. It was a light-staffing day and we responded as a three person engine company. As the senior firefighter I was the acting company officer and my new girlfriend at the time, who just happens to be my wife now, was the backseat firefighter. Get ready for the “Awwww” moment… it was our first fire “as a couple”. There was a number of cool things that came out of the fire, but one of them was the fact that Gina grabbed *my* maul.

On our main engine, there’s an 8-pound maul (big hammer) that I grab as my tool of choice every time I jump off the truck for a fire. It just tucks so neatly in my SCBA’s belt and is so compact yet handy that I make a beeline for it every time. This time, Gina had taken it, so I grabbed a pick-head axe.

It’s amazing when I have my maul how every access problem looks like something that I can solve by whacking it with a hammer of some sort. On this fire, I learned that when one has an axe, every problem looks like it can be solved by some sort of chopping.

Moral of the story, Gina and I entered the structure, saved the pooches, and stopped the fire in its trucks with minimal damage. There’s actually a hilarious video that I believe is still on our department’s web site that I’d let you see if I didn’t hide the name of the department(s) I work for due to reasons of wanting to remain employed.

And, like a lot of posts I write, I told you that so I could tell you this about an EMS call I responded to an indeterminate amount of time ago. I have the honor and privilege to be the senior medic on most shifts I work and I precept a lot of students on the ambulance. This shift was no different and this 0-dark-30 call illustrates a point that I’d like to explain to you.

For this call, the primary ambulance out of our station responded because they were on the way back from another call and my partner and I responded in our ambulance because we were up on the alternating call rotation. They arrived at the poorly-accessible apartment complex a few minutes before we did and made first patient contact. As it turns out, the middle age patient had ran out of his/her prescription Lasix (a potent diuretic, or water pill) a week or so prior to the call and had been retaining a great deal of excess bodily fluid. The patient’s legs were markedly and grossly swollen and weeping fluid out of fluid filled blisters. The Patient called us because he/she could no longer stand the pain of the cellulitis (infection) that had developed. The patient had no respiratory compromise, his/her lungs were clear, and he/she really had no other complaints. The patient had an extensive medical history of organ failure and disease. He/she was fully alert and oriented, and was able to assist us as we simply picked him/her up and carried him/her to the cot.

As we were loading the patient up in the ambulance and I was about to get into the back to continue my assessment and treatment of the patient, the EMT from the other ambulance who happens to be an almost-done Paramedic student told me, “So those legs are the worst I’ve ever seen fluid wise, you’re going to push some lasix on this one”. I mumbled something and got into the truck. I was tired and wasn’t really able to form complete sentences at the time due to sleep deprivation. I continued my assessment where I found that the frail patient had a blood pressure in the 70 systolic range (Low!) and that in addition to retaining fluid in his/her legs, he/she was also retaining fluid in his/her abdomen and was probably in need of a paracentesis. I managed the patient with a (beautifully executed, I must say) IV stick into an impossibly small and crooked vein, and gave just enough fluid to bring his/her BP up a bit without adding to his/her fluid overload all that much. I put the Pt on oxygen and a cardiac monitor, which revealed a normal sinus rhythm without ectopy and obtained a 12-lead EKG as well, which was not indicative of any acute problems. The patient stated that his/her pain was managed by padding and positioning of his/her swollen legs and even though he/she complained of no breathing problems, I put him/her on a bit of oxygen via nasal cannula.

The transport was uneventful, although his/her blood pressure never did come up. The ER later diagnosed the Pt with complete liver failure and toxicity.

But the interesting part of the story is this, when I got back the medic student asked me about giving IV lasix to the patient, as we carry that in our medication stock and have it available as an emergency diuretic for patients in congestive heart failure and/or fluid overload with pulmonary edema and respiratory compromise. He was almost taken aback when I said that I didn’t give any.

I asked him if he did a full assessment. He said that he had tried… but that he didn’t have enough time before I arrived and we took the patient out to the ambulance. I gave him my assessment findings and the news of the very low blood pressure. He said that he agreed with me on not giving the lasix with the markedly low blood pressure but was curious when I explained that it wasn’t the reason I didn’t give the medication.

We in EMS, and especially new providers carry our own hammers… our treatments and medications that we’re able to give in the field. Medics that use these treatments more often are called “aggressive” and it is a badge of honor. In fact, in some cases, aggressive field treatment is indeed warranted and improves patient outcomes. However, in a lot of cases it is not indicated and patients benefit from what we don’t do more so than from what we could have done.

This patient didn’t have any respiratory compromise and while he/she obviously could have benefited from the dieresis or removal of the excess fluid, she didn’t meet the criteria for emergent field administration of lasix, which is respiratory compromise from pulmonary edema. I made the decision to let the physician evaluate the patient and determine the best treatment path that would fit in with the patient’s ultimate plan of care. I didn’t believe that the patient would ultimately benefit from my administration of lasix twenty minutes earlier than the ER could have done it if the physician so chose.

Every treatment we administer must be given with a full assessment of the risks and benefits to the patient for doing so. Every EMS person should familiarize themselves with the long-term care paths of the conditions we treat and try to maximize the long-term benefit to the patient with the acute and short-term care we give. Not every problem is “a nail” and sometimes the hammers we carry aren’t the best ultimate solution for excellent patient care. Remembering how we as EMS people fit into the grand scheme of the overall healthcare system and in the ultimate care paths of our patients will help us all to do what we’re supposed to do, which is to provide excellent and appropriate patient care.

It is also of note, I guess, that Gina rarely steals my maul anymore. Now that we’re married… I “give it freely” to her.. What’s mine is her’s, as they say.

——————————————–
 
The original post has some pictures of the fire and of the doggies that my wife and I saved on our first “Fire Date” – It can be seen HERE

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Filed Under: Case Reviews, Clinical Discussion, Critical Judgment, Education, Medical Emergencies, paramedics, Pharmacology

67 y/o male CC: Syncope

05/17/2010 by Adam Thompson, EMT-P 12 Comments
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Also posted over at 12-Lead ECG Blog, go check out all the other great stuff there!

A 67 y/o male has fallen to the ground at his residence. His “partner” called 911 after seeing that he was unconscious. Upon your arrival the patient is alert and requesting that you pick him up because he really needs to make a bowel movement. The patient denies syncope but states that he does not remember falling.

Here is the initial ECG and the subsequent 12-lead ECG. Sorry for the poor quality.
What do you see?
What do you want to know?
What do you want to do?
****Update****

His Vital Signs

HR correlates with monitor, pulse not palpable at radial.

Initial BP 78/60

AAOx3, normal mental status, just wants to make a bowel movement.

Skin – Pale, more pronounced and white from the waste down. Skin was relatively dry.

****Update 5/20/2010****
A new 12-lead ECG is captured during transport.
The patient’s vital signs do not improve dramatically with IV fluids.
****Update 5/23/2010****
During transport the patient’s condition declined rapidly. After the 12-lead ECG above was captured, the patient went in to a decorticate posture. As most of you know, this is indicative of some sort of neuro compromise. With his airway control, mental status, and respiratory rate all declining, brainstem herniation was at the top of the list of differentials.
The patient became pulseless and apneic just prior to arriving at the ER–according to my partners, just after he released the bowel movement. The patient was not revived.
So what happened here?
Me and the field training officer came up with a few possible solutions. First, there is ST-elevation in the inferolateral leads of the initial 12-lead ECG. With the hypotension, a RCA occlusion is a possibility. If the patient has a dominant RCA, there appears to be some ST-depression in the septal leads, but this is a RBBB pattern, so with the T-wave discordance, the ST-depression is not a good clinical indicator of posterior wall involvement.
First Possibility: Right-sided infarct with hemodynamic compromise leading to a syncopal episode. The syncope caused a secondary head injury which cerebrally herniated during transport. I would like to note that this is highly unlikely. Also, the patient did not improve with fluids, which would have happened with a traditional RV infarct.
Second Possibility: It is much more likely that the patient had an atypical hemorrhagic stroke that presented with the first symptom of syncope. The changes on the 12-lead ECG could just be concurrent with cerebral ischemia. This is not completely understood, but theories involving nerve endings in the myocardium are abundant. The patient’s ICP would have increased during transport with the final result being cardiac arrest.
Third Possibility: Abdominal aortic aneurism with severe secondary cerebral ischemia due to hemodynamic instability. I’m not fond of this idea even though the AAA fit the picture in the beginning, it does not explain the decorticate posturing.
We also keep the huge possibility that we have no idea what happened on the list. Ok, so I wish I had more to give you, but an autopsy was not performed on this patient. It remains a mystery.
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Filed Under: Case Reviews, ECG/EKG Archive

Pucker Up: Bradycardia

05/13/2009 by Adam Thompson, EMT-P 12 Comments
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ECGs:


History of Present Illness:
Upon arrival the patient was laying on the walkway to the beach. FD stated that she was AAOx3. Her husband stated that she had a brief loss of consciousness. The patient denied any pain, but felt weak and short of breath. During transport the Zoll pacemaker was not functioning, it read “poor pad contact”. A new set of pads was tried without success. The patient’s perfusion status was rapidly declining, and we became unable to obtain a BP. Her ventricular rate slowed to about 10 BPM, and Epinephrine was given. The Epinephrine brought the HR up, Dopamine was then started. With the 3rd set of pacer pads the pacemaker began to get capture. The Dopamine was stopped after 1min. of administration. The patient’s perfusion status improved. Versed was administered for conscious sedation. Verbal report was given to the ER physician and care was transferred.

This was a call that I ran. The epi was a quick decision and luckily, it worked for as long as I needed it to. This wasn’t the most perfect scenario, but I improvised, adapted, and overcame the situation. Top ECG in first image shows initial rhythm, complete heart block. This is why Atropine wasn’t considered. Since Atropine Sulfate works on the vagus nerve, and the nerve is located in the atria, AV disassociation would impair the function of the med.
As you can see, the second strip, bottom of first image, and third strip on the top of second image are both post-epinephrine. Finally, the pacemaker was fixed and electrical capture is shown in the final strip. The patient’s pulse reflected full capture.
I would be happy never running a call like this again!
Just want to make it clear that this is not our protocol, and is not in any way endorsed by AHA. Read RM’s comment on administering Epinephrine to a cardiac patient. It is dangerous and was not something I wanted to do. At the moment, I felt the patient was going to code faster than I would have managed an infusion. Had I planned better and conquered the pacer problem faster, this could have been avoided.

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Filed Under: Case Reviews, ECG/EKG Archive

STEMI EKG & Angio

05/08/2009 by Adam Thompson, EMT-P Leave a Comment
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Prehospital 12 Lead:

Actual information from EMS patient care report:
Narative:
PT STATES HE WAS ASLEEP AND WAS WOKE OUT OF A SLEEP BY SEVERE SUBSTERNAL CHEST PAIN, 10/10 ON PAIN SCALE. PT STATES HE TOOK THREE OF HIS OWN NITROGLYCERIN AND CALLED 911. PT STATES AT TIME OF INITIAL EMS ASSESSMENT PATIENT’S PAIN WAS A 2/10 ON PAIN SCALE. PT DENIES NAUSEA, DIAPHORESIS, VOMITING, OR SHORTNESS OF BREATH. PT IS ALERT AND ORIENTATED. PT ADMITS TO DRINKING APPROX 5 ALCOHOLIC DRINKS THIS EVENING, AND STATES HE DID TAKE A VIAGRA THIS EVENING SEVERAL HOURS BEFORE BEDTIME. DURING INITIAL ASSESSMENT PATIENT STATES PAIN DID RE-OCCUR AND WAS NOW A 10/10, WITH RADIATION TO THE NECK AND JAW. PT IS STILL DENYING NAUSEA OR SHORTNESS OF BREATH.

Medical assessment:
Mental Status: Normal Mental Status for Patient, Oriented-Person, Oriented-Place, Oriented-Time, Oriented-Events, ; Neuro: Normal, ; Eyes: R: Reactive,; L: Reactive, ; Skin: Normal, ; Head/Face: Normal, ; Neck: Normal, ; LUQ: Normal (Soft, Non-Tender), ; LLQ: Normal (Soft, Non-Tender), ; RUQ: Normal (Soft, Non-Tender), ; RLQ: Normal (Soft, Non-Tender), ; GU: Normal, ; Cervical: Normal (No Pain or Deformities), ; Thoracic(back): Normal (No Pain or Deformities), ; Lumbar: Normal (No Pain or Deformities), ; Extremities: Upper R: Normal, ; Upper L: Normal, ; Lower R: Normal, ; Lower L: Normal, ;

Chest/Lungs: Chest Pain/Pressure (Non-reproducable), SEVERE PAIN 10/10 RADIATING TO NECK, AND JAW;

Vitals:
01:02 92/60 80 RR 20 Normal 97 Low O2 2/10 Right Arm (Supine)
01:12 88/60 78 RR 20 Normal 100 High O2 10/10 Right Arm (Semi-Fowlers)
01:24 86/56 72 RR 22 Normal 100 High O2 10/10 Right Arm (Supine)
01:31 118/63 72 RR 20 Normal 100 High O2 10/10 Right Arm (Trendelenburg)
01:38 108/60 68 RR 20 Normal 100 High O2 8/10 Right Arm (Trendelenburg)

Angiogram Pre & Post-treatment: Right Anterior Oblique View

Proximal LAD lesion resulting in decreased blood flow to anterior & septal walls.

ACS Feedback Form: Door to balloon time was 77 min.

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Filed Under: Cardiology, Case Reviews, ECG/EKG Archive

AMS Unknown Etiology

05/08/2009 by Adam Thompson, EMT-P 5 Comments
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I just thought I’d share this interesting call. I was pretty close to calling Dr. House.

Dispatch:

Medic 15 respond to 1212 Statin rd for a welfare check.

Now in case you don’t know what welfare checks are. They are for when you haven’t seen someone in a while, or you smell something funny coming from the neighbors house. Usually a welfare check ends up in two ways; either the resident is on vacation and no one is there, or dead upon arrival.

On Scene:
There is a lady standing outside the residence. She states that she comes to take care of the resident every day. She is positive that he is inside, and he can’t come to the door. She has called all the local hospitals, she has even called his favorite bar and his family members. I called our dispatch to make sure we haven’t responded to the residence recently, and we hadn’t. No one else has seen him.
The bystander states that the patient has a history of lung cancer. He is normally alert and oriented, and walks on his own.
So we try all the windows and door, we look for an extra key, but to no avail. We wait for law enforcement and decide it’s time to force entry. FD decides to take the door with the “K tool”. It’s basically a device that helps remove a deadbolt. After about 15 swings, and a t-shirt of sweat, the door is opened. We sent the deputy in first, of coarse. After all, he got the gun. Shortly after clearing the first floor, he goes up stairs, “you better get up here guys, he’s still breathing”.
Patient Contact:

A 67 y/o male was laying in left lateral recumbent position on the floor of his bedroom. There was no blood on the carpet or nearby walls. The patient was unresponsive with normal respirations. I initially suspected head-trauma from vertical deceleration*, and gave the patient a quick physical exam, or as my medic instructor would put it, “touchy feely”. I was thinking that maybe the caregiver was knocking on the door and when the patient got up to get it, fell and hit his noggin. The patient appeared to be atraumatic though, not even a scratch.
As I was near the patient’s head I noticed some audible “junk” in his lungs. This could just be from the cancer, but what’s a little oxygen going to hurt? He was sating around 95% before the mask, and 100% on it.
I also felt that the patient had a warm chest & belly, and cool extremities. I guessed this meant something neuro, at least that was what I thought I had read before.
Next, it was time to go through my AEIOU-TIPS, you know, that pneumonic for coma patients. After obtaining a normal blood sugar, and as I’m getting a temperature the patient quickly sits up with eyes wide open. I say “seizure” aloud and run downstairs to find out if he is epileptic. No dice, the patient has no history of seizures; it still wasn’t out of the question though.
We found Zocor, Coumadin, and some breathing treatments, no narcs or benzos.
So I get back up stairs and decide it’s time to get this patient ready for transport. The patient is completely aphasic(not talking) the entire time, I considered this a possible postictal state. At this point his vitals are as follows: BP 110/50, HR 100 & regular, SaO2 100%, BG 130, pupils equally reactive, and a temp of 100.4. The cardiac monitor showed a normal sinus rhythm(or sinus tach if you’re being specific). We had to utilize the stair-chair which was kind of difficult. The patient wasn’t being the most cooperative. He was acting like a combative hypoglycemic or head trauma; one that wouldn’t speak.
During Transport:

The patient didn’t become anymore alert in the back of the ambulance, so I obtained an IV, which took an extra person to hold the patient’s hands down. I then gave a slow administration of Narcan. This wasn’t the problem, and his respiratory status and pupils never really had me thinking it was. I also tried a stroke assessment but the patient wouldn’t follow my instructions. The 12-lead was unremarkable, and I was out of ideas.
Other than a cough here and there, the patient wouldn’t make a peep. At this point I was thinking maybe a CVA, electrolyte imbalance, meningitis, or encephalitis. His vitals remained stable and we just monitored him during transport. It was the weirdest thing, the patient was acting like he was just tired. He would roll over on to his left side and tuck himself in.
Transfer of care:

We dropped the patient off at the ER, and the nurse was as confused as I was. The patient’s aphasia was really making it hard on us. I mean, I have seen expressive aphasia, but this guy wasn’t even making a sound. He would look right at us and not acknowledge anything we said. He would just roll back over and tuck himself in.
Back at the station, me and the guys started placing bets on the cause.
I called back, about 2 hours later, the patient was diagnosed with rhabdomyolysis.
Where is that in the AEIOU-TIPS pneumonic?
We usually here about rhabdomyolysis when referring to compartment syndrome. It is by definition, the breakdown of skeletal muscle. This guy didn’t have any signs of compartment syndrome so I did some reading and found there are tons of causes of rhabdo. Simvistatin given with antibiotics can cause it. If he was taking his Zocor and some Amoxicillin, that could have been the cause.
I hate not knowing what’s wrong with the patient, even though it’s bound to happen from time to time. These are the calls that keep me reading. You better believe, the next call like this, rhabdomyolysis will at least be a consideration. Luckily, not knowing what was wrong with the patient didn’t hinder us from treating him. I’m not so arrogant as to think I will know it all, or I should know it all, just thought I’d share this call.
*vertical deceleration – Fall
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